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identified microRNA-155 as a potent driver of epileptogenic through PI3K/AKT/mTOR hyperactivation, revealing novel cross-talk between non-coding RNAs and canonical signaling pathways in neurological disease pathogenesis ( Figure 4 Moreover, PYC has been reported to enhance glucose uptake in a dose-dependent manner by promoting the membrane translocation of glucose transporter protein (GLUT4) and increasing the relative abundance of Akt mRNAs through the PI3K pathway (Lee et al., 2010)

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10.1016/S2468-1253(20)30084-4 8 Bellmann-WeilerR.LanserL.BarketR.RanggerL.SchapflA.SchaberM.et al (2020)

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